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Twenty patients died and 14 patients discontinued treatment due to non-virologic causes throughout the antiviral therapy course, none with SVR12 data available

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Data was self-reported by 162 surveyed customers who checked in with their provider on their second check-in between 4/12/222/8/26

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The first evidence that eNOS uncoupling may be a pathophysiologically relevant phenomenon was provided by in vitro studies in which it was demonstrated that native low-density lipoprotein 81 and even more pronounced oxidized low-density lipoprotein 82 are able to stimulate endothelial superoxide production and that this phenomenon is inhibited by the NOS inhibitor N G -nitro-L-arginine methyl ester, pointing to a causal role for eNOS in superoxide production

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