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Preliminary study on a pathogenic kinetoplastid protist in wild-captured Portunus trituberculatus

Moreover, following HQ treatment, N-acetylcysteine butyl ester (NACBE) protected RPE cells from cellcell junction disruption and, in general, ester derivatives of NAC prevented mitochondrial oxidative damage, measured as mitochondrial depolarization, even better than MitoQ, a well-known mitochondrial-targeted antioxidant

Usher syndrome type I G (USH1G) is caused by mutations in the gene encoding SANS, a protein that associates with the USH1C protein, harmonin

In some cases, symptoms do not appear until approximately 70% of the dopaminergic cells in the substantia nigra are already destroyed

Therapeutic modulation of the kynurenine pathway in severe mental illness and comorbidities: a potential role for serotonergic psychedelics

Cancers (Basel) 12 (8)