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Pharmacological inhibition of NOX using apocynin, a natural organic compound also known as acetovanillone [202] (Figure 3), decreased O 2 - levels and increased cell viability in MO59J human glioblastoma cells expressing mutSOD1 [114] and decreased ROS levels in primary astrocytes expressing mutSOD1, also restoring motor neuron survival in cocultured hESC-derived motor neurons with human primary astrocytes expressing SOD G37R [203] (Table 2)

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Mitochondrial transmission in AC neurons from the treated side to the contralateral (intact) side was also more obvious in the P-Mito group than in the Mito group (Fig

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