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10.1016/j.phymed.2023.155111 178 Quesada-VzquezS.BoneC.SahaS.TrigueroI.Colom-PellicerM.AragonsG.et al (2022)

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223 Notably, the interface between the ERAD and UPR also includes a negative feedback loop, exemplified by limiting IRE1 and ATF6 signaling upon their targeting for ERAD by HRD1

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Mitochondriatargeted antioxidants protect Friedreich Ataxia fibroblasts from endogenous oxidative stress more effectively than untargeted antioxidants