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F: Western blot for GSTP1 after immunoprecipitation with an anti-HPV-16 E7 antibody in control and HPV-16 E7-expressing HaCaT cells as well as in the CaSki and SiHa cell lines expressing endogenous HPV-16 E7

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BSO also significantly improved the decrease in mEPSC amplitudes by MeAIB, though to a smaller degree than its effect upon mEPSC frequency (Fig

34, 35, 36 We extend these findings in a model of chronic tobacco exposure, which indicate the presence of systolic function impairment and that arbiters of this response include molecular pathways of increased inflammation and oxidative stress