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Acacetin binds specifically to the ATP domain of HSP90, causing HSP90 to detach from COX-2, which leads to COX-2 being ubiquitinated and broken down in macrophages, thereby reducing inflammatory reactions (189)

doi: 10.1016/j.cmet.2020.06.010 28 MiyajimaM

To sustain the genome stability, cells have evolved a complicated fine-tuning mechanism, termed as DNA-damage response (DDR), to detect and repair DNA lesions

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The change in peak area should have a linear relationship with the mass concentration of glutathione, with a goodness of fit (R) reaching 0.9995 or above