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But as mentioned earlier consult your doctor first

Ataxia-telangiectasia mutated gene controls insulin-like growth factor I receptor gene expression in a deosyribonucleic acid damage response pathway via mechanisms involving zinc-finger transcription factors Sp1 and WT1

Consequently, there is an increase in intestinal permeability and translocation of intestinal flora, leading to the release of enterogenic endotoxins into the bloodstream which act on the original organ via the circulatory system, further exacerbating deterioration of the intestinal organ