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This could suggest an overactive Gln-Glu cycle in adults with ASD, which may represent either a manifestation of, or a compensatory response to, dysregulated excitatory neurotransmission implicated in the pathophysiology of ASD

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Fath, T

As almost all TyrA sequences within clade II have a non-acidic residue (Gln or Asn) at the corresponding 222 position, except for Chloroflexi TyrAs (Figure 2, Figure S1), they are likely prephenate-specific TyrA p enzymes

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[88, 92] have documented the identification of species-specific RNA A-to-I editing sites in the RDL of insecticide-resistant mosquitoes