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Emodin and AZT synergistically inhibit the proliferation and induce the apoptosis of leukemia K562 cells through the EGR1 and the Wnt/-catenin pathway

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Subsequent remodeling of the growth and fibroblast subtypes effectively inhibits scar formation

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Concluding remarks The fundamental processes of ferroptosis, such as lipid peroxidation and GPX4/GSH system functions, have been studied for a long time 15,19,154

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Edelstein, S

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body weight: 0.13, 95% CI 0.34, 0.55)

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generated experimentally using directed evolution as descried herein) for desired targeting of various tissues

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