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The mechanism is straightforward your immune system has been generating free radicals at high rates, and your antioxidant reserves are lower than usual

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Furthermore, oxidative stress contributes to neuronal damage and mediates neuroinflammation, blood-brain barrier (BBB) disruption, and the production of spasminogen following SAH ( CU has demonstrated multiple significant neuroprotective effects, particularly in conditions such as ischemic stroke, traumatic brain injury, and intracranial hemorrhage (Thiyagarajan and Sharma, 2004)

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