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XuJZhangXYanLZhangZWeiJLiLet alInsight into lotusine and puerarin in repairing alcohol-induced metabolic disorder based on UPLC-MS/MS

[405] report an 88% regressive ASD phenotype in a systematic investigation of infants with and without a family history of ASD

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A systematic review and meta-analysis further confirmed these findings (137)

BPC-157 at 0.0045 mg/lb, 0.0000045 mg/lb, or even 0.0000000045 mg/lb considerably inhibited this syndrome, particularly the hyperthermia and wet dog shake components strongly associated with 5-HT2A overactivation

In summary, development defects in the CNS in GDH-HI patients could be caused by multiple factors, including chronic hyperammonemia, and hyperinsulinemia, the more detailed mechanisms need to be further studied