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Because GHK-Cu delivers Cu to tissues, large or repeated parenteral doses could in principle contribute to copper accumulation in individuals with impaired copper homeostasis (for example Wilson disease)

Research has demonstrated that the DM-associated protein CD36 promotes substantial release of arachidonic acid and PGE2 by modulating ERS-dependent calcium influx, thereby triggering inflammatory cascades (Kuda et al., 2011)

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HDPs are the leading cause of maternal and perinatal morbidity and mortality, especially in early-onset and underserved areas

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