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Using external samples from the ADNI study, we demonstrated that our MRS was significantly associated with disease progression (i.e., from CN to MCI/AD, or MCI to AD), after accounting for covariates age, sex, APOE 4 status, education, baseline diagnosis, and baseline MMSE scores

In BNIP3-mediated mitophagy in mammals, ULK1 phosphorylates BNIP3, enhancing its interaction with LC3 and reducing BNIP3 proteasomal degradation of 271,272 (Fig

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Atg16L1/AP2/clathrin heavy chain interactions contribute to the formation of plasma membrane autophagosomes