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Subsequent investigations demonstrated that FSP1, regulated by Nrf2, significantly inhibited lung cancer cell growth and sensitised Keap1 -deficient cells to ferroptosis by knockdown or inhibition of FSP1 expression in Keap1 post-mutant lung cancer [46, 78]

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The researchers concluded that epithalon may prolong the lifespan of cell populations through anti-senescence activity [2]

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Typically, the incidence and seriousness of the toxicity is proportionately related to the concentration of the drug in the body and to the duration of the exposure