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Notably, p38 MAPK is highly sensitive to oxidative signals, with low-dose H 2 O 2 sufficient to induce its rapid activation in muscle cells, preceding activation of catabolic regulators like FoxO and NF-B ( Once activated, p38 MAPK and JNK promote transcription of muscle atrophy-related genes

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lncRNAs, long non-coding RNAs

This is likely attributable to the extensive signal crosstalk and intricate feedback regulatory networks within the PI3K pathway, which render sustained anti-tumor effects difficult to achieve with single-target inhibition

[DOI] [PubMed] [Google Scholar] 87.Winnike JH, Li Z, Wright FA, Macdonald JM, OConnell TM, et al