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Therefore, activation of autophagy following epilepsy could be a compensatory mechanism to limit neuronal loss

Notably, some pain-relieving drugs also act via additional signaling pathways

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Temporary relief of pain after administration of these 2 drugs further solidifies the diagnosis of trigeminal neuralgia

both antibiotic use and germ-free conditions suppress the expression of CYP7A1 and CYP27A1, key enzymes involved in BA production based on animal mechanistic studies (Chiang and Ferrell, 2019)