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A quintessential example is the burgeoning body of evidence demonstrating that reactive oxygen species (ROS) can serve as triggers for diverse forms of cell death collectively known as oxidative cell death

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This makes it preferable for studies examining anabolic signaling, muscle protein synthesis rates, or recovery speed from tissue damage

We consider that the reduced Glrx expression and Grx1 activity in the mouse lungs is one of the mechanisms of protection against oxidative stress