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J Ethnopharmacol 301:115819

further research is needed to enable broader applicability in the case of breast cancer (120)

However, this preservation of mitochondrial function creates a paradox: by maintaining the electron transport chain, CHAC1 inadvertently supplies the ROS that propagate iron-dependent lipid peroxidation ( via two convergent mechanisms: the direct depletion of glutathione and the indirect facilitation of mitochondrial ROS production, thereby coupling sustained metabolic activity to ferroptotic cell death under cysteine limitation

We review risks, unknowns, alternatives, and contraindications before considering treatment

[oai_citation:10PubMed]( Tendon explant/fibroblast studies: enhanced explant outgrowth, stress survival, and migration

Targeting astrocyte-mediated glutamate transport (e.g., enhancing GLT-1 function) or lactate shuttling (e.g., activating MCT1/2 transporters) represents a promising therapeutic strategy to preserve neuronal bioenergetics and slow disease progression (Yang et al., 2024)