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While these compounds and mechanisms serve to maintain hormone homeostasis, they significantly limit the usefulness of peptides in a therapeutic sense

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Once adapted, TAMs themselves become major ROS producers, primarily through NOX2 activity, using ROS to suppress cytotoxic T cells and NK cells [147, 148]

IECs necrosis has long been associated with the severity of IBD inflammation, and abnormal IEC death can impair intestinal barrier function, exacerbating inflammatory responses [59]

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