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There is a very strong body of experimental evidence indicating a neuroprotective role of UA, mediated through anti-inflammatory and antioxidative mechanisms that themselves are likely to be closely interrelated (summarised in Fig

What Pickart's body of work establishes: GHK-Cu is a biologically active molecule with characterized effects on fibroblast behavior, collagen synthesis, and copper-dependent enzymatic pathways in vitro

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Base excision repair and nonhomologous end joining are involved in repairing these damages 3,4

Ajoene was reported to augment the therapeutic effects of cytarabine and fludarabine in resistant myeloid leukemia cells, by enhancing caspase-3 activation and Bcl-2 inhibition (52)