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APS enhances autophagosome formation and LC3-I conversion to LC3-II by activating the PI3K/AKT/mTOR signaling pathway while down-regulating p-AKT and p-mTOR and up-regulating PTEN, thereby inhibiting the over-activation of the PI3K/AKT/mTOR pathway

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Specialized niches like the perivascular and endosteal niches help sustain DTC dormancy through direct and indirect interactions with host cells

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A later dose-response study explored the relationship between T4 dose and functional recovery after stroke, reinforcing that the effect is dose-dependent rather than all-or-nothing