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216 Ferroptosis in metabolic disorders As a key regulation mechanism in metabolic diseases, the imbalance of glucose and fatty acid metabolism can directly or indirectly participate in ferroptosis and influence its occurrence and development

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In studies of human glioblastoma, the absence of exogenous Gln significantly reduces glutamate levels, resulting in the conversion of the dimer GLS1 into a self-assembled, extremely low Km filamentous polymer that further depletes intracellular Gln

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