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Lipid peroxidation produces damaging byproducts such as malondialdehyde (MDA) and 4-hydroxynonenal (HNE), which impair membrane integrity and worsen neurological and cardiovascular disorders

However, cytosolic H 2 O 2 generated by APx1 deficiency can trigger the adaptation to oxidative stress [27]

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Finally, we outline future directions within an ethnopharmacological perspective, integrating traditional knowledge with pharmacological innovation to inform multi target, standardized, and potentially personalized interventions for defined ASD endophenotypes

HDL has antioxidant properties that safeguard LDL from oxidative damage and help eliminate oxidized lipids, thereby inhibiting the generation of intracellular ROS