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GSTP1 acts as a direct inhibitor of JNK in vivo to regulate constitutive expression of specific downstream molecular targets of the JNK signaling pathway (161)

Sci Adv 6(31):eaba0466

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and reduce stiffness in the arteries of patients with type 2 diabetes

This apparent paradox arises from their context-dependent roles: cell death in normal intestinal cells may disrupt mucosal homeostasis, thereby aggravating inflammation and CD pathogenesis, whereas selective elimination of damaged cells via these pathways might suppress disease progression and synergize with other therapies