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In the early stage, a large number of studies focused on tumor cells, but with the continuous understanding of tumor structure, TME has been proved to play a key role in the process of tumor occurrence, development, metastasis, drug resistance, and the acquisition and maintenance of tumor characteristics depend on the role of TME to varying degrees (11, 12)

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Beyond psoriasis, FABP5 inhibition could be explored in other ferroptosis-linked epithelial disorders such as atopic dermatitis or radiation-induced dermatitis, where oxidative lipid damage undermines barrier integrity

Under pathological conditions such as oxidative stress, hyperactivation of PARP-1 significantly promotes the accumulation of -syn (Puentes et al., 2021)

On the other hand, pathophysiological features exhibited the association between mitochondrial dysfunction, decreased activity of complex I and complex IV of the electron transport chain, and the glutathione system in MS [23]