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The methylglyoxal pathway supports growth of Salmonella on glucose Having established the activation of the methylglyoxal pathway in Salmonella following oxidative stress, we determined if this pathway participates in the growth of Salmonella in glycolytic substrates

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FIGURE 1 2.1.2 Genetic predisposition GWASs have identified multiple gout-associated genetic loci, revealing a dual-pathway urate metabolism-inflammatory regulation genetic architecture

Under physiological conditions, a healthy gut microbiota and an intact intestinal barrier are critical for establishing immune tolerance ( The tumor immune microenvironment (TIME), a dynamic ecosystem of cancer cells, stromal cells, ECM, and diverse immune cells, profoundly shapes tumor progression and therapeutic responses ( A pivotal mechanism linking gut dysbiosis to pro-tumorigenic inflammation is the translocation of microbial components, most notably lipopolysaccharide (LPS), across a compromised intestinal barrier ( Beyond TLR-dependent microbial sensing, the NF-B pathway serves as a critical signaling hub that integrates diverse pro-tumorigenic stimuli within the TIME

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