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tenuifolia and exhibits various pharmacological effects, such as antioxidant [12], anti-inflammatory [13], inhibition of neuronal apoptosis [14], and enhancement of neuroprotection [15]

doi: 10.1042/bj2060267 17 ShibuyaNKoikeSTanakaMIshigami-YuasaMKimuraYOgasawaraYet al

MLKL drives basal resistance to ferroptosis by depleting PUFAs, whereas ACSL4 promotes basal resistance to necroptosis by making the membrane less susceptible to MLKL-driven permeabilization [16, 214]

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