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Kowalczyk, E

In PD, the M1 phenotype may be induced by -syn aggregates, signaling through Toll-like receptors (TLR), releasing cytokines and neurotoxic molecules that promote cytotoxic responses, leading to persistent neuroinflammation (Li et al., 2021a)

Values are the average of three independent experiments performed in triplicateSEM

56 ZimmermannP.LauleO.SchmitzJ.HruzT.BleulerS.GruissemW

MYC mediates PHGDH activation, which enhances GSC self-renewal by regulating redox homeostasis, promoting on-carbon metabolism and facilitating the DNA damage response via SSP activation, thereby driving GSC malignant progression and radioresistance in GBM (64)