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This effect was not sensitive to naloxone, indicating a distinct mechanism from its opioid-mediated antinociceptive effects 8

The mechanism by which thiol-binding metals exert a toxic activity on cells and organisms is in general terms described as the consequence of interaction with and inhibition of essential thiol groups of enzymes and proteins, although the systemic toxicity of the metals is very different [2]

No clear changes in plasma ChE activity or other parameters such as reaction time, airway resistance, or vision were found

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Third, immunometabolic crosstalk in the regulation of ferroptosis highlights the pivotal role of chronic low-grade inflammation in OA pathogenesis

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