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In order to obtain sufficient fusion protein with high activity, the optimal inducible expression conditions for TAT-CcSOD1 were explored

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[] Greater glutamate-glutamine and N-acetylaspartate concentrations and reduced myo-inositol concentrations suggest the patient is in the NAC therapy arm, according to MR spectroscopy of certain groups in that research

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Sorafenib induces ferroptosis by targeting the cystine/glutamate anti-transport system Xc- [154,155,156,157], an effect that impinges on cystine uptake, thereby preventing subsequent synthesis of glutathione (GSH), the major intracellular antioxidant, preventing GPX4 activation [158, 159], and can trigger ER stress and ferroptosis