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The ground-breaking finding that mice expressing dominant-negative N-cadherin, an adherent junction protein, develop spontaneous (not induced using chemicals or infections known to induce colitis in mice) inflammation in the gut epithelium further supports the key role of cell-to-cell adhesion, especially in its capacity to contribute to overall mucosal barrier function in regulating mucosal immune homeostasis

Increasing the SC dose to compensate for the lower bioavailability leads to unpredictable serum concentrations and can therefore not be recommended

74 Dysfunctional telomeres arise from critically short telomeres, ultimately leading to replicative cellular senescence and chromosome instability: both hallmarks of aging

though superficially similar to those used for gaming devices, they are not optimized for real-time control and therefore can usually accept input from only one direction at a time

All animal studies were approved by the local animal welfare authorities (Regierung von Unterfranken) with the animal protocol no

Always start with low doses and observe for any adverse reactions