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Ernst C, Nagy C, Kim S, Yang JP, Deng X, Hellstrom IC, et al

Copper overload stimulates lipoylated protein aggregation and subsequent ironsulfur cluster protein loss through direct binding to lipoylated components in the tricarboxylic acid (TCA) cycle, resulting in proteotoxic stress and subsequent cell death ( Growing evidence indicates potential crosstalk between cuproptosis and periodontitis

These potential synergies indicate that H could be strategically integrated as an adjunct to existing treatment regimens, enhancing neuroprotection and functional outcomes

This metabolic shift aligns with ferroptosis research wherein 5-HT and 3-HAA promote tumor cell resistance to ferroptosis

The clinical field covers weight management, energy, cognitive performance, skin health and recovery

Han YC, Lim Y, Duffieldl MD, Li H, Liu J, Abdul Manaph NP, et al