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A deletion polymorphism in glutathione-S-transferase mu (GSTM1) and/or theta (GSTT1) is associated with an increased risk of toxicity after autologous blood and marrow transplantation

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IRF3 inhibits nuclear translocation of NF-B to prevent viral inflammation

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In addition, this deletion completely cancels the increase in ROS production caused by the gmhA deletion , while all other mutations in the genes that regulate the metabolism of cysteine and glutathione, as well as a violation of cystine transport, lead to an even greater increase in the level of ROS in cells with a gmhA deletion

Neurometabolite changes in response to antidepressant medication: a systematic review of 1H-MRS findings