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The involvement of stress response genes in regulating apoptosis in HOSC were confirmed through higher expression levels of stress response genes such as SOD3 (FC=4.66, qvalue =0.0141) and GPX3 (FC=6.81, qvalue =0.0006) suggesting the role of stress response genes in rescuing HOSC from undergoing apoptosis in spite of disruptive mitochondrial OXPHOS

Glutathione Injection Site Reactions: Reaction Type Comparison Understanding reaction severity and timeline helps differentiate manageable inflammation from complications requiring intervention

Amelioration of bromobenzene hepatotoxicity by Withania somnifera pretreatment: role of mitochondrial oxidative stress

This enhanced blood flow facilitates nutrient delivery and waste removal at injury sites, accelerating the healing process

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