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Furthermore, the extent of modification of the putative regulatory thiol by persulfidation needs to be quantified, as low stoichiometries of modification can only be regulatory if they activate a gain-of-function of the modified protein

257 Taken together, these observations suggest that cancer cells acutely exposed to CDDP might temporarily rely on glycolysis as a source of energy (presumably reflecting the mitochondriotoxic effects of cytoplasmic CDDP), whereas the acquisition of stable CDDP resistance might require a robust respiratory activity

NAD+ MOTS-c is a small peptide produced inside your mitochondria (the parts of your cells that generate energy)

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