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Recently, a study further revealed that IGF1-induced BNIP3 expression requires NRF2 to act through downstream transcriptional factors HIF-1 and NRF1 (Riis et al., 2020)

A separate 2025 narrative review by McGuire and colleagues asked a blunter question in its title: "Regeneration or Risk?" [2] Their conclusion: the preclinical evidence is consistently positive, but the absence of human data makes it impossible to generate evidence-based practice guidelines

First, the molecular trigger specificity of oxalate-induced ferroptosis in renal tubular cells is undefinedhow oxalate precisely initiates the synergistic dysregulation of iron uptake (TFR1), lipid metabolism (ACSL4), and antioxidant defense (GPX4/SLC7A11) remains elusive, with no clarity on upstream signaling molecules or post-translational modifications that link oxalate exposure to ferroptosis machinery

Similarities in B cell defects between aging and obesity

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Comparative analysis reveals a role for TGF- in shaping the residency-related transcriptional signature in tissue-resident memory CD8+ T cells