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When ROS accumulation exceeds the tipping point, their carcinogenic effects on proliferation and development are shifted to antitumour effects via the induction of programmed cell death (PCD) [47, 49], including apoptosis, autophagy, necroptosis, pyroptosis, and ferroptosis

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injections with a dose of 100 nmol kg 1 of the experimental compounds

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This makes stem cell therapy more likely to take [4]