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doi: 10.1016/j.cgh.2023.04.015 3

In particular, Y185 ECL2 and H186 ECL2 act as built-in agonist for activating GPR52, thus resulting in a high level of basal activity of the receptor 50 (Supplementary Fig

The current treatment for COVID-19 ARDS is a combination of anti-viral and anti-inflammatory medications

(31), where higher L-carnitine concentrations in the medium inhibited -butyrobetaine hydroxylase activity but left -N-trimethyllysine hydroxylase activity unaffected