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A complicating factor in the concept of homosynaptic plasticity is the nature of input-specificity. Conventionally, plasticity is called input-specific if no changes are observed in an independent test input, not stimulated during the induction, i.e., no heterosynaptic changes

Avoid scratching, which can introduce bacteria and lead to secondary infection

Liver biopsies of PBC show portal-based inflammation with bile duct injury (see LFN Post 7)

Goldstein and colleagues described it as a component of the thymosin fraction from calf thymus, and later work demonstrated its ubiquitous expression in nucleated mammalian cells and high concentration in platelets [1]

The transamination reaction depends heavily on the conversion of pyridoxine, also known as pyridoxol and gravidox, to pyridoxal phosphate (PLP)