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& Blasiak, J

This complement-dependent mechanism represents the primary pathogenic cascade responsible for ultrastructural damage at the neuromuscular junction in AChR-positive MG

Adolescent patients with non-seminomatous GCTs had slightly improved survival compared with adults, despite presenting with more advanced disease, achieving excellent survival outcomes [288]

Plant Biotechol

Lipid-engorged foamy macrophages that accumulate after traumatic SCI depend on PI3K activity to phagocytose myelin debris while simultaneously suppressing autophagic clearance of lipid droplets

These cells formed 3D spheres, which were embedded in Matrigel (Corning Inc.) and cultured in hIO medium [Advanced DMEM/F-12, 2% B-27, 100 ng/mL EGF, and 100 ng/mL Noggin]