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Limitations of human biomonitoring and objectives HBM relies not only on validated analytical tools, but also on a clear understanding of the relationship between exposure to a toxin and concentration of that toxin (or metabolite) in a biological sample

Dysfunction of glucose metabolism impairs the normal functioning of neurons, which is widely observed in neurodegenerative diseases [17]

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Thus once HO enzymes were activated by curcumin, it led to subsequent activation of ARE and Nrf2 pathways to neutralize both the oxidative and nitrosative stresses [271, 272]

[Google Scholar] Potential risk modifications of GSTT1, GSTM1 and GSTP1 (glutathione-S-transferases) variants and their association to CAD in patients with type-2 diabetes

GSH is the most abundant antioxidant and is essential in maintaining cellular homeostasis