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Ferroptosis-associated oxidative stress also influences post-translational modifications of ATG proteins, fine-tuning their activity and localization (63-65)

Complement activation is a critical pathway in the pathogenesis of inflammatory skin diseases such as acne, yet its specific mechanisms of action and key regulatory factors remain to be elucidated

Works to neutralize free radicals in the body causing oxidative stress to the cellular structure

Penetrating 10 layers deep into the skin, its lightweight, non-sticky texture ensures swift action in reducing pigmentation

Antioxidant treatment prevents serum deprivation- and TNF--induced endothelial dysfunction through the inhibition of NADPH oxidase 4 and the restoration of -oxidation

In this study, we first demonstrated that ferroptosis occurred locally at the injury site of TSCI, and was closely associated with lipid metabolism and the PPARa signaling pathway