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Wang L , Wheeler DA

Gene alterations modulating glutamine dependency in cancer The dependency of glutamine metabolism in cancer is not simply a passive adaptation to proliferative state, but rather a tightly regulated response to a multitude of intracellular and extracellular factors, including potential genetic alterations

Antibodies are often purified based on the highly specific interaction that occurs between an antibody and its antigen (the sequence recognized by the antibody)

During the progression of neurodegenerative disorders, astrocytes respond to chronic inflammation and pathogenic stimuli by acquiring distinct reactive phenotypes that act through non-cell-autonomous mechanisms to influence neuronal and glial homeostasis 4

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