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Figure 2 3.2 B cell metabolism supports autoantibody production and intrarenal pathogenesis in lupus nephritis B cells drive SLE pathogenesis by differentiating into autoreactive plasma cells that produce pathogenic autoantibodies, acting as antigen-presenting cells to activate autoreactive T cells, and secreting pro-inflammatory cytokines, thereby collectively promoting immune complex deposition, end-organ damage, and systemic autoimmunity (112)

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Translational efforts face an additional barrier: while CSF1R inhibitors allow experimental microglial manipulation, recreating their physiological complexityincluding precise spatial distribution and functional heterogeneitypresents a significant challenge for therapeutic development