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A Phase I Study of FT538, a First-of-Kind, Off-the-Shelf, Multiplexed Engineered, iPSC-Derived NK Cell Therapy As Monotherapy in Relapsed/Refractory Acute Myelogenous Leukemia and in Combination with Daratumumab or Elotuzumab in Relapsed/Refractory Multiple Myeloma

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Research suggests it may act through beta-3 adrenergic pathways or through non-receptor-mediated effects on adipocyte lipid metabolism, rather than through classical GH receptor signaling

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Similar to the NNMT knockdown experiments, NNMT inhibitors reversed obesity by modulating NAD + rescue and the pathways mediated by SAM

Possible contributing factors include bacterial or parasitic infections, immune system dysregulation and food intolerances or allergies, particularly to specific proteins