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5 Conclusion Using a CCl 4 -induced acute liver injury model, this study demonstrated that GGV provides protective effects, as evidenced by improvements in ALT and AST levels and histopathological outcomes in mice

Its activation promotes the release of proinflammatory cytokines such as 1 L-1 and IL-18 and induces pyroptosis, leading to endothelial injury and TF release, thereby fueling thrombo-inflammation

It can be speculated that the PI3K/Akt/mTOR signaling pathway may lead to LPS-induced intestinal barrier damage through autophagy and regulatory protein synthesis

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These GTE polysaccharides suppressed the intracellular levels of reactive oxygen species (ROS), as well as elevating antioxidative enzymes including superoxide dismutase (SOD), catalase (CAT), glutathione peroxidase (GPx) and glutathione (GSH)