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The study posits that DSIP's actions on substance P, beta-endorphin, and corticosterone levels are part of a broader spectrum of biochemical changes, hinting that DSIP might initiate a series of molecular reactions contributing to its stress-modulatory actions

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This might be reflected in the observed changes in glycolysis pathway metabolite levels in both our NSCLC HIF1 KO model and NSCLC patient blood samples in response to radiotherapy treatment