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These compounds can serve as the potential biomarkers of lipid peroxidation [5, 35]

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Acta Pharmacol Sin [Internet]

3d), while the AMPAR antagonist CNQX (which prevents BiC/4-AP-induced bursting that leads to NMDAR activation) did inhibit protection (Supplementary Fig

Collectively, our data suggest that the PARG inhibitor PDD promotes targeted degradation of BRD2/3/4 via enhanced BRDPROTACCRL2 VHL ternary complex formation and K29/K48-linked ubiquitylation