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I would tend to agree with you Having spent an unhealthy amount of time reading scientific and advocate material about b12 deficiency and it's treatment it very largely does not align with the Google answer quoted

Moreover, gut microbes produce neuroactive compounds such as SCFAs and tryptophan metabolites that influence brain function and renal physiology

Mass spectrometry identity MS confirms the molecular weight matches the target sequence exactly proving the compound IS what the label says, not a misfold or substitution

Hydroxocobalamin is often the first choice in clinical B12 therapy due to its extended duration in the body

In more detail, the FKBP12-rapamycin complex binds to the FRB domain of mTORC1 to produce steric hindrance, which interferes with interactions between the substrate and the active site of mTORC1 [15, 16]

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