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b) deficit secondari di Carnitina in pazienti con acidurie organiche su base genetica tipo propionic acidemia, metil-malonic aciduria, isovaleric acidemia ed in pazienti con difetti genetici della beta-ossidazione

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These findings suggest that the pathophysiology associated with the loss of FMRP or MeCP2 may not arise from irreversible developmental brain abnormalities, but result from functional disturbances of neural circuits that could be corrected in adulthood, providing a potential rational basis for treatment of neurodevelopmental disorders in adulthood (Castrn et al., 2012